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This review was limited by a dearth of studies demonstrating the effect of supplementation on the time taken to achieve ketosis as defined by the lingua franca of NK, ≥0.5 mmol L −1 and on symptoms of keto-induction during this time. Research performed on exogenous ketone supplements is, at this time, highly preliminary, and has been predominantly performed using animal subjects. Ketone ester supplements significantly elevated BOHB levels for the duration of the 4-week study. Similarly, there is a paucity of research on the short-chain fatty acids and their effects on ketogenesis. While the ketogenic amino acid leucine may not independently encourage ketogenesis to levels consistent with NK, more research is required, and the effect on time to NK and symptoms of keto-induction, particularly in a classic KD, are at this stage unknown. It has, for example, been observed that mood is improved within the first two weeks of a diet irrespective of macronutrient composition (Rosen et al., 1985), and only one study, to our knowledge, has demonstrated a correlation between ketone levels and memory performance (Krikorian et al., 2012). It’s unclear at this time whether an elevation in ketones over and above NK would mitigate the effects of keto-induction. So, it is more accurate to say that exogenous ketones mimic the effects, many of which are positive, of NK, rather than inducing it. While it is clear that exogenous ketones increase serum BOHB, they are not ketogenic, and may, in fact, inhibit endogenous ketone production (Balasse & Neef, 1975). Both ketone esters and salts elevate BOHB to levels consistent with NK (Holdsworth, Cox & Clarke, 2016), with ketone esters having greater effects on ketonaemia with ketone salts providing significantly higher reporting of gastrointestinal symptoms (Stubbs et al., 2016). 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