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It is unclear whether a genetically tractable model that recapitulates PCOS can be created, given that humans are usually monoovulatory, whereas mice are polyovulatory. Consistent with an increased risk of ovarian cancer in women with endometriosis, mice with both of these genetic changes develop and die from uterine endometrial ovarian cancer. Advanced knowledge of these pathways may aid in diagnosis and treatment of implantation failure in women. These progesterone receptor–Cre mice helped to define essential roles of BMP2, chicken ovalbumin upstream promoter transcription factor II, steroid receptor coactivator 2 (SRC2), PTEN and mitogen-inducible gene-6 in uterine biology and cancer and the ovulatory function of peroxisome proliferator–activated receptor-γ. Beginning his career in emergency medicine, Dr. Moghadam developed a deep understanding of patient safety, anatomy, and medical aesthetics. 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