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This occurs in spite of the ability of hemoglobin-injected i.c.v. or in the PVN to abolish/reduce the increase in NO metabolites in the male rat PVN extracellular dialysate induced by the above substances [79,80,81,96,104] or when this increase occurs in physiological contexts, [e.g., in the presence of a sexually receptive female rat (non-contact erections) or during copulation] (Table 4), or when induced by oxytocin injected into the caudal ventral tegmental area as well (Table 9). Further experiments are required to verify if guanylate cyclase is involved in the facilitatory effect of oxytocin injected in the ventral subiculum and to ascertain if mechanisms similar to those summarized above are also operative in the posteromedial cortical nucleus of the amygdala and in the bed nucleus of the stria terminalis of male rats after the injection of oxytocin at doses that increase NO production and induce penile erection [49,151,152]. At variance from the PVN, the available data suggest that NO, produced by the activation of NO synthase located in the cell bodies of mesolimbic dopaminergic neurons by the increased Ca2+ influx that takes place after activation of oxytocinergic receptors expressed in these cell bodies by oxytocin, activates guanylate cyclase in the ventral tegmental area of male rats in order to facilitate penile erection induced by oxytocin injected into this mesencephalic area [47,48] (Table 9). However, these studies suggest that guanylate cyclase, one of the best-known targets of NO in peripheral tissues and in several brain areas as well [5,11,12,25,157] is not involved in the facilitatory effect of NO on erectile function in the PVN. 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