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Hoorn and colleagues observed no difference between fasted and non-fasted ketogenic protocols for time to ketosis, without specifically describing their definitions for ketosis or the time to ketosis itself (Kang et al., 2007; Chul Kang et al., 2005). This treatment for epilepsy was pioneered at Johns Hopkins University Hospital (Livingstone, 1972; Livingston, Pauli & Pruce, 1977), and is referred to as a ‘classic’ or ‘standard’ ketogenic diet. To elicit this carbohydrate restriction, while also providing sufficient alternate fuel to ensure sustainability of the diet, i.e., in comparison to fasting to achieve ketosis, VLCKDs have been used to encourage ketosis. Ketogenic diets elicit a state of ketosis known as ‘nutritional ketosis’ (NK), a state of hyperketonaemia distinct from pathological ketosis such as diabetic ketoacidosis (DKA) (Krebs, 1966). There is limited research on the potential for other supplements to improve time to NK and reduce symptoms of keto-induction. There is a clear ketogenic effect of supplemental MCTs; however, it is unclear whether they independently improve time to NK and reduce symptoms of keto-induction. Animal studies indicate that the short chain fatty acids acetic acid and butyric acid, increase ketone body concentrations. Keto gummies, like most supplements, exist in a space where marketing often outpaces scientific evidence. Response to keto gummies varies significantly between individuals. Some products marketed as “keto-friendly” may contain ingredients that function similarly to sugar in the body, such as maltodextrin, dextrose, or maltitol. There is no universal dosage for keto gummies, as formulations vary significantly between products. As always, it’s advisable to consult healthcare professionals when making significant changes to your nutrition or supplement intake. 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A 6‐month study in subjects with metabolic syndrome showed that an American Heart Association Step 2 diet was ineffective for sustained improvement glycemic control, but when psyllium (3.5 g twice a day before meals) was added to the controlled diet, fasting blood glucose, insulin, and HbA1c were all significantly reduced (Figure 1; Cicero et al., 2010). While postprandial glucose studies are useful for assessing the acute glycemic effects of fiber, longer (multimonth) intervention studies are needed to determine if a gel‐forming fiber can provide a clinically meaningful improvement in glycemic control in patients at risk for, or being treated for, type 2 diabetes mellitus. Nonviscous soluble fiber supplements (e.g., inulin, wheat dextrin, partially hydrolyzed guar gum) and insoluble fiber (e.g., wheat bran) do not provide this gel‐dependent beneficial effect (McRorie & McKeown, 2016). 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